Introduction to diabetes mellitus

Diabetes
mellitus is a metabolic disorder whereby there is increase blood sugar
considered to be normal for FBS 65-110mg/dl for RBS65mg-140mg. Diabetes
mellitus is characterized by abnormally high level of sugar (glucose in the
blood).

When
the amount of the glucose in the blood increases e.g. after a meal, it triggers
the release of hormone insulin from the pancreas. Insulin stimulates muscle and
fat to remove glucose causing the blood sugar level to increase to normal
levels.
Conditions
where the blood sugar level consistently run too high is diabetic and it is the
most common endocrine disorder. Diabetes has a potential long term complication
that can affect the kidney, eye, heart, blood vessel and nerves.
All
patients suffering from diabetes likely tend to be grouped with diabetes, the
truth is that there are two different types of diabetes which are similar in
their elevated blood sugar, but different in many ways.
When
the topic pertains to both types of diabetes, we use the general term
“diabetes”. Diabetes is correctly divided into two major sub-groups: type 1
diabetes and type 2.
This
division is based upon whether the blood sugar problem is caused by insulin
deficiency (type 1) or insulin resistance (type 2). Insulin deficiency means
there is not enough insulin being made by the pancreas due to a malfunction of
their insulin producing cells. Insulin resistance occurs when there is plenty
of insulin made by the pancreas (it is functioning normally and making plenty
of insulin) but the cells of the body are resistance to its action which result
in the blood sugar being too high.
Gestational
diabetes is a term used to describe the condition in which a non-diabetic woman
has higher than normal blood sugar during pregnancy. For enlightenment of
causes of diabetes, unfortunately knowledge is scanty. But some factors
relating to inheritance and over nutrition which may explain why some people
develop diabetes and other do not.
0 0 votes
Article Rating
Subscribe
Notify of
guest

0 Comments
Inline Feedbacks
View all comments
0
Would love your thoughts, please comment.x
()
x